Jump to content
swole troll

Controlling E2... It's actually a little difficult

Recommended Posts

15 hours ago, swole troll said:

Entirely variable depending on individual and the PEDs being used 

With dianabol and test suspension being the fastest ways to bring it back up outside of exogenous estradiol use.

So I’m on 180 mg test e and I was on 12.5 aromosin twice a week and running both for around 12 weeks but I stopped aromosin around 26 days ago. I aromatise quite easily even on 125 m test e I will be above normal medichecks eostrogen ranges. I’ve got morning erections but poor libido.

 

cheers

Share this post


Link to post
Share on other sites
7 hours ago, gavzilla said:

So I’m on 180 mg test e and I was on 12.5 aromosin twice a week and running both for around 12 weeks but I stopped aromosin around 26 days ago. I aromatise quite easily even on 125 m test e I will be above normal medichecks eostrogen ranges. I’ve got morning erections but poor libido.

 

cheers

Without bloodwork it's just a guess 

So you're assuming your e2 is high right now? 

Logic would dictate that is your issue 

But again without bloods and feeling it out we won't know.

This can be a problem with running supraphysiological dosages between cycles

You end up spinning plates. 

Share this post


Link to post
Share on other sites
15 hours ago, swole troll said:

Without bloodwork it's just a guess 

So you're assuming your e2 is high right now? 

Logic would dictate that is your issue 

But again without bloods and feeling it out we won't know.

This can be a problem with running supraphysiological dosages between cycles

You end up spinning plates. 

I do normally get bloods done but I can’t right now.

i will presume after 26 days absent of aromosin running 180 mg test e that my eostrogen would have recovered by now.

Cheers for the reply 

Share this post


Link to post
Share on other sites

Thanks @swole troll - the information here is Great. Thanks a lot for posting!!

Bit of a complex area controlling E2. I’ve heard alot about raloxifine over nolva. el chapo or ghost.recon in the AMA said it’s more effective than nolva, also implying ralox + arimidex as the  “go to” on cycle method of controlling E2. What are your thoughts? 

As I understand it: arimidex to lower e2... ralox to prevent/reduce gyno... So I should have ralox on standby, but only necessary to Run it at first signs of any symptoms? and Many are able to control e2 with just an AI? 

I’m currently on 175mg Sustanon TRT + 1000iu HCG weekly. E2 was 140pnmol on last blood test but is creeping upwards. I’m wanting to add 125mg test E in so I’m running  300mg “test” total (as a first cycle)... and I’m wondering 0.5mg armidex EOD will suffice?  

Thanks!

Share this post


Link to post
Share on other sites
1 hour ago, Pez189 said:

Thanks @swole troll - the information here is Great. Thanks a lot for posting!!

Bit of a complex area controlling E2. I’ve heard alot about raloxifine over nolva. el chapo or ghost.recon in the AMA said it’s more effective than nolva, also implying ralox + arimidex as the  “go to” on cycle method of controlling E2. What are your thoughts? 

As I understand it: arimidex to lower e2... ralox to prevent/reduce gyno... So I should have ralox on standby, but only necessary to Run it at first signs of any symptoms? and Many are able to control e2 with just an AI? 

I’m currently on 175mg Sustanon TRT + 1000iu HCG weekly. E2 was 140pnmol on last blood test but is creeping upwards. I’m wanting to add 125mg test E in so I’m running  300mg “test” total (as a first cycle)... and I’m wondering 0.5mg armidex EOD will suffice?  

Thanks!

Well it's two different pathways of preventing gynecomastia formation or enlargement. 

raloxifene and tamoxifen are SERMs, selective estrogen receptor modulators 
and their MOA is to bind to the receptor in the nipple in order to prevent oestrogen from doing so and causing glandular growth (with raloxifene being the more potent binder of the two) 

arimidex is an aromatase inhibitor, it's MOA is to deactivate the aromatase enzyme so that it cannot convert testosterone into oestrogen, subsequently lowering oestrogen levels which then prevents gyno formation 

two different approaches, both of which simply limiting the amount of oestrogen binding to the breast site in one way or another.

So yes combination treatment works and is often used in those with breast cancer.

The only thing worth noting is that tamoxifen increases the speed at which the liver metabolizes arimidex and letrozole causing a reduction in efficacy by 27 and 38% respectively so perhaps your AI isn't lowering your e2 to quite the level you are used to when using X dose however your gyno is still controlled (largely by the SERM) but blood work will still be something worth looking at should other elevated e2 symptoms arise. 

There is no interaction at all between aromasin and tamoxifen or raloxifene 

and to my knowledge there is no interaction between raloxifene, arimidex and letrozole

only tamoxifen, arimidex and letrozole. 

to your last question I don't know. you could aromatase heavily, you could respond well to AI's or equally you could respond poorly to AIs 

I can't give a cookie cutter answer, just a case of suck it and see 

also anything under 400mg isn't worth the shut down, I don't care what anyone says and they're free to start a thread and tag me in it if they wish to discuss it further (do not clutter this one with debate on this) but yes 175mg is a generous TRT. 

Share this post


Link to post
Share on other sites

Create an account or sign in to comment

You need to be a member in order to leave a comment

Create an account

Sign up for a new account in our community. It's easy!

Register a new account

Sign in

Already have an account? Sign in here.

Sign In Now

×