Quote:
Originally Posted by ah24  Increases mitochondria production too. . |
increased mitochondria production by acute exercise leads to damaged muscle and other tissues by an increased oxidative stress on the electron transport associated with the mitochondria.
During exercise the increased electron flow through the mitochondrial electron transport chain leads to an increased rate of ROS production (reactive oxgen species related to muscle/tissue damage) which is known to cause excessive production of free radicals
The isolation of mitochondrial fractions by differential centrifugation has shown that the amount of damaged mitochondria, recovered in the lightest fraction, is remarkably increased by long-lasting high intense exercise (own study)
low/mod intensity over a controlled peroid of time leads to the increase in mitochondrial density and increases in the maximal activities of a number of mitochondrial enzymes of the TCA cycle (tricarboxylic acid cycle) and oxidative pathways
Exercise at a given oxygen uptake, show there is less of a decrease in the high-energy phosphates, a smaller increase in creatine and ADP in after training results and this is believed to provide a reduced stimulus to glycogenolysis and glycolysis and increase the reliance on fat catabolism during exercise.